It’s uncharted waters to be honest. We know there are many viruses that can cause myocarditis, most of them very common. Just a rare complication of some viruses that cause pink eye, the GI bug or the common cold. The problem with myocarditis is that once you get it the course can be variable from severe acute congestive failure and deadly arrythmias to reversible diminished function and no residual effects to anything in between. We don’t have enough data to determine if covid induced myocarditis causes a more concerning or less concerning long term picture. That’s the issue here, ERod is literally in uncharted medical territory, which isnt where you want to be as a mid twenties professional athlete.
There are two pieces of info I’d like to know that haven’t been released.
1. Has he had any arrythmias? Myocarditis can lead to ventricular and atrial dysrhythmias and if he’s had ventricular dysrhythmias, then he’d be a candidate for a defibrillator.
2. What is his residual Ejection fraction? The standard person ejects 60-65% of the blood that enters the left ventricle with each beat. People with myocarditis can get all the way down to 5% and be in transplant range. Recovery can be variable, but does happen in most cases, but if his EF is down, then that means he’s at a higher risk of developing heart failure under stress or generating dysrhythmias which would keep him from playing.
Without the above two questions, everything is speculation. So for now, you just wait to see if he gets cleared. If he’s not cleared early on, that tells you there is residual dysfunction, and if he still has dysfunction many months out, his likelihood of recovery back to normal would be low